How GVT Works
GVT, a drug made from Monosodium Luminol, has the power and capacity to treat many neurodegenerative diseases, specifically ALS (Lou Gehrig's Disease) & Gulf War Illness (GWI), but has remarkable potential to also treat a wide range of other diseases, including Parkinson's, Alzheimer's, Huntington's, Multiple Sclerosis (MS) and Ataxia-Telangiectasia (A-T).
All these neurodegenerative diseases share many common traits as they all affect the neurons in the human brain. In effect, the brain needs to maintain a "redox balance" (also called an oxidation-reduction reaction). Neurodegenerative diseases throw off this balance and cause cell damage.
Here's where GVT steps in: GVT acts as a buffer that restores the redox balance, then reverses and stops the damaging process. The regulation of the redox state is vital for cell vitality, activation, and even organ function.
When the redox balance does not remain within the careful bounds normal for the body, it leads to oxidative stress, mitochondria impairment, and inflammation; all of which lead to neural cell death.
An imbalance in the redox state can arise both naturally in the body, as well as from environmentally-induced conditions such as radiation exposure or even cigarette smoke. In the case of GWI - often this imbalance was set in progress by chemicals and agents introduced to soldiers during wars and conflicts around the world. DEET, Agent Orange, and overwhelming amounts of mental stress are all believed to be root causes of GWI and redox imbalance.
GVT acts as the redox buffer the body can not always produce. A buffer helps to resist the pH change and neutralizes the levels of pH in the body, keeping things stable. Buffer solutions are key in biology and medicine because most reactions, like the redox reactions that occur in our bodies, need to remain between a very specific pH range to work properly.
Our drug GVT not only acts as the buffer, but it stops or even reverses many of the steps between environmental stress and revives redox-suppressed cellular defenses.